Thymosin β4 improves functional neurological outcome in a rat model of embolic stroke

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Thymosin beta4 improves functional neurological outcome in a rat model of embolic stroke.

UNLABELLED Thymosin beta4 (Tbeta4) is a developmentally expressed 43-amino acid peptide that inhibits organization of the actin-cytoskeleton by sequestration of G-actin monomers. Tbeta4 improves cardiac function after myocardial infarction in adult mice and promotes healing properties in both dermal and corneal wounds. We tested the hypothesis that Tbeta4 improves functional neurological outcom...

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Thymosin β4 Improves Neurological Outcome and Enhances Induced Oligodendrogenesis in the Rat after ICH

Thymosin β4 (Tβ4), a G-actin binding protein, has diverse biological functions. This study tested the effects of Tβ4 on oligodendrogenesis in a rat model of intracerebral hemorrhage (ICH). ICH was induced by stereotactic injection of 100 μm of autologous blood into the striatum in 32 male Wistar rats. The rats were randomly divided into four groups: 1) saline control group (n = 8); 2) 3 mg/kg T...

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Induced pluripotent stem cells (iPSC) constitute a powerful tool to study cardiac physiology and represents a promising treatment strategy to tackle cardiac disease. However, iPSCs remain relatively immature after differentiation. Additionally, engineered heart tissue (EHT) has been investigated as a therapy option in preclinical disease models with promising results, although their vasculariza...

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Thymosin β4 Accelerates Wound Healing

Craniofacial Developmental Biology and Regeneration Branch, National Institute of Dental and Craniofacial Research, NIH, Bethesda, Maryland, U.S.A.; *Center for Combat Casualty and Life Sustainment Research, Department of Pathology, USUHS, Bethesda, Maryland, U.S.A.; †Department of Biochemistry and Molecular Biology, The George Washington University School of Medicine and Health Sciences, Washi...

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Preventive antibacterial treatment improves the general medical and neurological outcome in a mouse model of stroke.

BACKGROUND AND PURPOSE Epidemiological studies have demonstrated a high incidence of infections after severe stroke and their prominent role in morbidity and mortality in stroke patients. In a mouse model, it has been shown recently that stroke is coupled with severe and long-lasting immunosuppression, which is responsible for the development of spontaneous systemic infections. Here, we investi...

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ژورنال

عنوان ژورنال: Neuroscience

سال: 2010

ISSN: 0306-4522

DOI: 10.1016/j.neuroscience.2010.05.017